| Allegra Dosages & Strengths | ||||
|---|---|---|---|---|
| Strength | Format | Route | Strength | Class |
| Allegra 180 mg | tablet | oral | 1.0 each | OTC |
| Allegra 30 mg | tablet | oral | 1.0 each | OTC |
| Allegra 60 mg | tablet | oral | 1.0 each | OTC |
| Allegra 60 mg | capsule | oral | 1.0 each | OTC |
| Allegra 30 mg/5 mL | suspension | oral | 5.0 milliliter(s) | OTC |
Posted by sherry327 24 days ago
I have lost a significant amount of weight since I began taking Allegra D two years ago. I struggled with my weight since hav...
Posted by sprine 5 months ago
I started taking Allegra about 2 weeks ago. It has definitely helped my seasonal allergies however, I have been very irritab...
Posted by vburrows 8 months ago
I have taken Allegra-D for a year now and have noticed extreme mood changes. I have even lost motivation for daily duties. I...
Hervey Bay ‘May Day’ stay pay offer - e-Travel Blackboard (press release)
Each Grand Mercure Allegra apartment makes the most of its superb position, with alfresco terraces and folding tropical shutters taking full advantage of ...
Sun Mar 09 18:19:39 -0400 2008
Allegra Diagnostics tests out $4M first VC round - Bizjournals.com
Allegra Diagnostics, a diagnostics startup with ties to Boston University, has reeled in its first round of venture capital financing to develop its ...
Fri Mar 07 10:18:46 -0500 2008
Ask Allegra: Easter - The Observer
Allegra McEvedy will be live online at 3pm on Wednesday March 12 to answer your seasonal cooking queries. Post your questions now! Easter is almost upon us. ...
Mon Mar 10 11:30:45 -0400 2008
fexofenadine hydrochloride - The hydrochloride salt form of fexofenadine, a carboxylated metabolic derivative of terfenadine and third generation selective histamine H1-receptor antagonist with antihistaminic and non-sedative effects. Fexofenadine competitively binds peripheral H1-receptors, thereby stabilizing an inactive conformation of the receptor. Consequently histamine binding and activity as a result of mast-cell degranulation followed by the release of multiple inflammatory mediators, such as interleukins, prostaglandin and leukotriene precursors, is blocked, thereby preventing the triggering of pro-inflammatory pathways.