Alprazolam, also known under the trade names Xanax (not to be confused with Zantac), Xanor, Alprax, and Niravam, is a short-acting drug of the benzodiazepine class. It is primarily used to treat moderate to severe anxiety disorders and panic attacks, and is used as an adjunctive treatment for anxiety associated with moderate depression. It is also available in an extended-release form, Xanax XR, both of which are now available in generic form. Alprazolam possesses anxiolytic, sedative, hypnotic, anticonvulsant, and muscle relaxant properties.
| Alprazolam ER Dosages & Strengths | ||||
|---|---|---|---|---|
| Strength | Format | Route | Strength | Class |
| Alprazolam ER 2 mg | tablet, extended release | oral | 1.0 each | OTC |
| Alprazolam ER 0.5 mg | tablet, extended release | oral | 1.0 each | OTC |
| Alprazolam ER 3 mg | tablet, extended release | oral | 1.0 each | OTC |
| Alprazolam ER 1 mg | tablet, extended release | oral | 1.0 each | OTC |
- Dop kun ned hunden om nødvendig - Østlandets Blad
- Dop kun ned hunden om nødvendigØstlandets BladAlprazolam er det beroligende middelet som ser ut til å ha best dokumentert effekt mot akutt angst hos hund, sier veterinær Nils Søli, ...and more »
Wed Dec 30 09:09:21 +0000 2009
Heath Ledger Post-Mortem: When Mixing Medicine Kills - Wall Street Journal
In 2005, nearly 600000 people showed up at the ER because of side effects from nonmedical use of prescription drugs — and most of those people had taken ...
Thu Feb 07 08:50:32 +0000 2008
Accidental Overdose Killed Heath Ledger - Guardian Unlimited
... temazepam, alprazolam and doxylamine as causing the actor's death. There was no mention of alcohol or illegal drugs.er she arrived for an appointment at ...
Wed Feb 06 20:34:28 +0000 2008
alprazolam - A triazolobenzodiazepine agent with anxiolytic, sedative-hypnotic and anticonvulsant activities. Alprazolam binds to a specific site distinct from the inhibitory neurotransmitter gamma-aminobutyric acid (GABA) binding site on the benzodiazepine-GABA-A-chloride ionophore receptor complex located in the limbic, thalamic and hypothalamic regions of the central nervous system (CNS). This binding causes an allosteric modification of the receptor and enhances the affinity of GABA to the receptor leading to an increase in the frequency of chloride-channel opening events. This leads to an increase in chloride ion conductance, neuronal hyperpolarization, inhibition of the action potential and leads to a decrease in neuronal excitability.