Benadryl Severe Allergy & Sinus Headache Information

What's inside Benadryl Severe Allergy & Sinus Headache

Benadryl Severe Allergy & Sinus Headache in the News

Peanut allergies hitting toddlers - Monterey County Herald
Until then Burks suggests keeping an antihistamine such as Benadryl on hand in case of emergency. Serious reactions typically require treatment with ...
Mon Dec 10 04:40:18 -0500 2007

A very restricted diet - St. Petersburg Times
Not quite sure why her daughter was having such unusual reactions, her mother, Missy Echevarria, gave her some Benadryl and immediately took her to a doctor ...
Wed Dec 12 01:18:56 -0500 2007

It's a mystery: Food allergies on the rise - Pueblo Chieftain
Seafood and peanuts seem to trigger the most severe food allergies - those causing anaphylaxis, a sometimes fatal allergic response. ...
Wed Nov 14 02:41:38 -0500 2007

Benadryl Severe Allergy & Sinus Headache Chemical Information

acetaminophen - An acetanilide with analgesic and antipyretic activity. Although the exact mechanism through which acetaminophen exert its effects has yet to be fully determined, this agent probably acts by inhibiting prostaglandin synthesis in the central nervous system (CNS). Inhibition of prostaglandin synthesis in the CNS results in elevation of the pain threshold and lowering of the thermal set point in the hypothalamus.

diphenhydramine hydrochloride - The hydrochloride salt form of diphenhydramine, an ethanolamine and first-generation histamine antagonist with anti-allergic activity. Diphenhydramine hydrochloride competitively blocks H1receptors, thereby preventing the actions of histamine on bronchial smooth muscle, capillaries, and gastrointestinal (GI) smooth muscle. This prevents histamine-induced bronchoconstriction, vasodilation, increased capillary permeability, and GI smooth muscle spasms.

pseudoephedrine hydrochloride - The hydrochloride salt form of pseudoephedrine, a phenethylamine and an diastereomer of ephedrine with sympathomimetic property. Pseudoephedrine hydrochloride displaces norepinephrine from storage vesicles in presynaptic neurones, thereby releasing norepinephrine into the neuronal synapses where it stimulates primarily alpha-adrenergic receptors. It also has weak direct agonist activity at alpha- and beta- adrenergic receptors. Receptor stimulation results in vasoconstriction and decreases nasal and sinus congestion.