Contac Day and Night Allergy Information

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Contac Day and Night Allergy in the News

The End of Antibiotics and the Rise of Iodine as an Effective ... - Natural News.com
"We know that antimicrobial resistance will follow antimicrobial use as sure as night follows day," said Dr. John A. Jernigan, deputy chief of prevention ...
Mon Mar 10 10:37:48 -0400 2008

Have the Last Laugh on Allergies This Season: Actress Molly ... - Earthtimes
WASHINGTON, Pa., March 5 -- With the first day of spring only 15 days away, actress and entertainer Molly Shannon is teaming with the Asthma and Allergy ...
Wed Mar 05 09:16:39 -0500 2008

Community Calendar Feb. 29-March 14 - Mansfield News
Fourth Tuesday of month during school year, 7-9 pm Support group for licensed day care providers. Contact Jane at 508-285-0019 for information. ...
Thu Feb 28 15:29:49 -0500 2008

Contac Day and Night Allergy Chemical Information

acetaminophen - An acetanilide with analgesic and antipyretic activity. Although the exact mechanism through which acetaminophen exert its effects has yet to be fully determined, this agent probably acts by inhibiting prostaglandin synthesis in the central nervous system (CNS). Inhibition of prostaglandin synthesis in the CNS results in elevation of the pain threshold and lowering of the thermal set point in the hypothalamus.

diphenhydramine hydrochloride - The hydrochloride salt form of diphenhydramine, an ethanolamine and first-generation histamine antagonist with anti-allergic activity. Diphenhydramine hydrochloride competitively blocks H1receptors, thereby preventing the actions of histamine on bronchial smooth muscle, capillaries, and gastrointestinal (GI) smooth muscle. This prevents histamine-induced bronchoconstriction, vasodilation, increased capillary permeability, and GI smooth muscle spasms.

pseudoephedrine hydrochloride - The hydrochloride salt form of pseudoephedrine, a phenethylamine and an diastereomer of ephedrine with sympathomimetic property. Pseudoephedrine hydrochloride displaces norepinephrine from storage vesicles in presynaptic neurones, thereby releasing norepinephrine into the neuronal synapses where it stimulates primarily alpha-adrenergic receptors. It also has weak direct agonist activity at alpha- and beta- adrenergic receptors. Receptor stimulation results in vasoconstriction and decreases nasal and sinus congestion.