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Flonase Information

What's inside Flonase

Flonase Active Ingredients: fluticasone propionate, details.
Flonase Dosages & Strengths
Strength Format Route Strength Class
Flonase 0.05 mg/inh spray nasal 1.0 inhalation OTC

Recent Flonase Side Effects

Posted by dustin 9 months ago
Sense of smell is lost.

Posted by idreamofjeanne about 1 year ago
It made me bloated, excitable, moody, depressed, angry and most of all, lost my sense of smell. Not to mention, 2 weeks after I stared taking it I caught a horrible cold and I never get sick, accept for my allergies. I only took this medicine for about 3 weeks. I cannot wait until it is out of my system. I know it has steroids in it and they are powerful drugs and it can stay in your system for a long time.

Posted by italia2 about 1 year ago
I have a 3 year old that was taking Singulair until today after I read the horrible side effects that I was seeing in my child. He is also taking Flonase, one squirt in each notstril every morning. Are there any concerns I need to know? Is this truly safe for a 3 year old?

Flonase in the News

AMITYVILLE, NY--(BUSINESS WIRE)--Hi-Tech Pharmacal Co., Inc ... - Business Wire (press release)
Mr. Seltzer further commented, “We are very up-beat about our generic business due to our recent approval of generic Flonase®*, and more recently, ...
Mon Mar 10 08:05:07 +0000 2008

Hi-Tech Pharmacal says milder winter hurt drug sales - Newsday
In January, Hi-Tech won final approval from the US Food and Drug Administration for a new nasal spray, Flonase, and the company said in its news release ...
Mon Mar 10 09:09:08 +0000 2008

Don't marry him! - San Diego CityBEAT
Tonight, Sam and I stood in the kitchen, each ingesting two squirts per nostril of our own individually marked bottles of Flonase. ...
Tue Mar 04 21:57:24 +0000 2008

Flonase Chemical Information

fluticasone propionate - The propionate salt form of fluticasone, a synthetic trifluorinated glucocorticoid receptor agonist with antiallergic, antiinflammatory and antipruritic effects. Binding and activation of the glucocorticoid receptor results in the activation of lipocortin that in turn inhibits cytosolic phospholipase A2, which triggers cascade of reactions involved in synthesis of inflammatory mediators, such as prostaglandins and leukotrienes. Secondly, mitogen-activated protein kinase (MAPK) phosphatase 1 is induced, thereby leads to dephosphorylation and inactivation of Jun N-terminal kinase directly inhibiting c-Jun mediated transcription. Finally, transcriptional activity of nuclear factor (NF)-kappa-B is blocked, thereby inhibits the transcription of cyclooxygenase 2, which is essential for prostaglandin production.


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