| Lidex Dosages & Strengths | ||||
|---|---|---|---|---|
| Strength | Format | Route | Strength | Class |
| Lidex 0.05% | solution | topical | 100.0 milliliter(s) | OTC |
| Lidex 0.05% | ointment | topical | 100.0 gram(s) | OTC |
| Lidex 0.05% | cream | topical | 100.0 gram(s) | OTC |
| Lidex 0.05% | gel | topical | 100.0 gram(s) | OTC |
Posted by lizz8 over 4 years ago
My family doctor gave this to me, to heal sores inside my mouth. The medication I got is Lidex gel. It does heal canker sor...
Medicis Reports Fourth Quarter and Year End 2007 Financial Results - Primenewswire (press release)
... SOLODYN(R) (minocycline HCl, USP) Extended Release Tablets, TRIAZ(R) (benzoyl peroxide), LIDEX(R) (fluocinonide) Cream, 0.05%, VANOS(R) (fluocinonide) ...
Wed Feb 27 16:13:32 -0500 2008
Medicis and Revance Announce Strategic Collaboration - CNNMoney.com
... TRIAZ(R) (benzoyl peroxide), LIDEX(R) (fluocinonide) Cream, 0.05%, VANOS(R) (fluocinonide) Cream, 0.1%, SYNALAR(R) (fluocinolone acetonide), ...
Tue Dec 11 16:52:51 -0500 2007
Medicis to Present At RBC Capital Markets Healthcare Conference - CNNMoney.com
... TRIAZ(R) (benzoyl peroxide), LIDEX(R) (fluocinonide) Cream, 0.05%, VANOS(R) (fluocinonide) Cream, 0.1%, SYNALAR(R) (fluocinolone acetonide), ...
Mon Dec 10 08:07:12 -0500 2007
fluocinonide - A synthetic glucocorticoid and derivative of fluocinolone acetonide with anti-inflammatory and antipruritic activity. Fluocinonide binds the glucocorticoid receptor, followed by translocation of the ligand-receptor complex to the nucleus and transcription activation of genes containing glucocorticoid-responsive elements. Lipocortin-1 is one factor induced by fluocinonide that interacts and inhibits cytosolic phospholipase 2 alpha, thereby preventing phospholipase translocation to the perinuclear membrane and subsequent release and conversion of arachidonic acid to inflammatory prostaglandins. In addition, MAPK phosphatase 1 is induced, thereby preventing the triggering of the MAPK cascade resulting in pro-inflammatory effects via Jun N-terminal kinase and c-Jun. Finally, fluocinonide binds to and inhibits nuclear factor kappa-B directly, resulting in inhibition of cyclooxygenase 2 transcription and subsequent prostaglandin synthesis.