Xanax XR Information
Xanax XR
Alprazolam, also known under the trade names Xanax (not to be confused with Zantac), Xanor, Alprax, and Niravam, is a short-acting drug of the benzodiazepine class. It is primarily used to treat moderate to severe anxiety disorders and panic attacks, and is used as an adjunctive treatment for anxiety associated with moderate depression. It is also available in an extended-release form, Xanax XR, both of which are now available in generic form. Alprazolam possesses anxiolytic, sedative, hypnotic, anticonvulsant, and muscle relaxant properties.
What's inside Xanax XR
Xanax XR Active Ingredients: alprazolam, details.| Xanax XR Dosages & Strengths | ||||
|---|---|---|---|---|
| Strength | Format | Route | Strength | Class |
| Xanax XR 3 mg | tablet, extended release | oral | 1.0 each | OTC |
| Xanax XR 2 mg | tablet, extended release | oral | 1.0 each | OTC |
| Xanax XR 0.5 mg | tablet, extended release | oral | 1.0 each | OTC |
| Xanax XR 1 mg | tablet, extended release | oral | 1.0 each | OTC |
Xanax XR in the News
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Wed Dec 23 22:47:31 +0000 2009
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Wed Jan 16 05:37:21 +0000 2008
FDA OKs Impax Anxiety Drug - TheStreet.com
The drug is marketed as Xanax XR by Pharmacia and Upjohn. Impax said it plans to launch the product soon. Shares of Impax, which trade on the Pink Sheets, ...
Tue May 29 13:21:01 +0000 2007
Xanax XR Chemical Information
alprazolam - A triazolobenzodiazepine agent with anxiolytic, sedative-hypnotic and anticonvulsant activities. Alprazolam binds to a specific site distinct from the inhibitory neurotransmitter gamma-aminobutyric acid (GABA) binding site on the benzodiazepine-GABA-A-chloride ionophore receptor complex located in the limbic, thalamic and hypothalamic regions of the central nervous system (CNS). This binding causes an allosteric modification of the receptor and enhances the affinity of GABA to the receptor leading to an increase in the frequency of chloride-channel opening events. This leads to an increase in chloride ion conductance, neuronal hyperpolarization, inhibition of the action potential and leads to a decrease in neuronal excitability.
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